UVM study uncovers cellular switch that lets H3N2 flu invade lungs
TL;DR Summary
A University of Vermont lab found that the Rab11B protein is essential for H3N2 influenza to enter human lung cells, challenging the idea that sialic acid is the universal entry point and suggesting new antiviral targets as H3N2 relies on Rab11B while H1N1 does not.
- One UVM lab’s surprise discovery led to flu science breakthrough VTDigger
- New ways to prevent flu revealed in 'accidental' lab breakthrough, study finds Fox News
- Bruce ‘Accidental’ Breakthrough Reveals How Influenza Viruses Replicate Within Cells, Breeze Reports University of Vermont
- UVM research reveals a new potential path to flu treatment and prevention New Hampshire Public Radio
- Human Lung Cell Infected With Influenza (IMAGE) EurekAlert!
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